Problem 1: Could you at any point make sense of why a reduction in serum lipid levels follows an intense myocardial dead tissue (MI)? How long does this decrease last, and in this case, how is it determined whether the patient has hyperlipidemia?
Problem 2: Why should aspirin be chewed instead of taken orally in patients who have had a myocardial infarction?
Problem 3: If the heart rate falls below 60 beats per minute, should beta-blockers that are prescribed for hypertension be stopped?
Problem 4: How can patients with recurrent ischaemic stroke be evaluated for aspirin resistance? Is the length of time it takes for the blood to clot an issue? Cardiovascular disease 13 133
Problem 5: Will a patient with heart failure and atrial fibrillation on warfarin who experiences frequent transient ischaemic attacks benefit from having aspirin prescribed? Does the substitution of aspirin with clopidogrel resolve this issue?
Problem 6: Is anticoagulation necessary for 70% of patients with inoperable carotid stenosis?
Problem 7: In the event of renal damage caused by hypertension, what is the upper limit of serum creatinine above which thiazides should not be prescribed?
Problem 8:
a. What is the measurement of blood pressure?
b. When a patient is suspected of having coarctation of the aorta, where should the stethoscope and cuff be placed?
Problem 9: As a third-year clinical understudy in Russia I'm exploring 'the job of proteolytic chemicals and their inhibitors in lung pathology'. Can you describe the normal function of proteolytic enzymes in the lung?
Problem 10: One of the causes of bronchial breath sounds in Question 2 is bronchtasis. It's hard to comprehend this. Could you provide a more precise explanation of how bronchial breath sounds work?