Problem: Alexandra, a 27-year-old bartender, was diagnosed with ADHD at age 10. She has been on and off medication since then, first on immediate-release methylphenidate, then on the methylphenidate patch. She experimented with illicit drugs during her late adolescence and is still a heavy drinker. After a few years of self-medication with alcohol and cigarettes, she is seeking medical attention again. You decide to put her on 80 mg/day of atomoxetine, one of the non-stimulant medications effective in ADHD. Why does atomoxetine lack abuse potential? A. It decreases norepinephrine levels in the nucleus accumbens, but not in the prefrontal cortex B. It increases dopamine levels in the prefrontal cortex, but not in the nucleus accumbens C. It modulates serotonin levels in the raphe nucleus D. It increases dopamine levels in the orbitofrontal cortex and anterior cingulate cortex. Need Assignment Help?